BibTex Citation Data :
@article{JKLI5962, author = {Nikie Dewanti and Onny Setiani and Nurjazuli Nurjazuli}, title = {Hubungan Paparan Merkuri (Hg) Dengan Kejadian Gangguan Fungsi HatiPada Pekerja Tambang Emas di Wonogiri}, journal = {Jurnal Kesehatan Lingkungan Indonesia}, volume = {12}, number = {1}, year = {2013}, keywords = {}, abstract = { Background: Traditional gold mining activities that using WOA/ amalgamation can cause Hg emissions to the environment. The emissions could raise mercury poisoning in the environment and human. Liver as a major part of the metabolism and accumulation of Hg in the human body, so that Hg could lead to liver damage. In the previous research, Hg exposure in male rats caused hepatotoxicosis. Average blood mercury levels of workers was 53.5 μg/m 3 . Objective: To determine the associa tion between mercury (Hg) exposure and the occurance of liver dysfunction on gold mine workers at Jendi Village, sub-district Selogiri, Wonogiri District. Methods: It was a cross-sectional study, total sample were 41 workers. The data obtained from the results of laboratory tests of blood samples and the results of the interview respondents.Data would be analyzed using biavariate and multivariate statstic test. Results: There was 41.16% of respondents we re miners, processors as well as grates, the average of working period wa s 10 years, work duration 6 hours a day and 6 days a week. 97.56% of respondents have blood mercury levels above normal (U.S. EPA: 5.8 ppb). Elevated levels of SGOT experienced by 24.4% of respondents , SGPT 17.1% of respondents and ALP 58.8% of respondents or as much as 68.3% of respondents having liver disfunction. There wa s no difference incidence of liver dysfunction seen from the type of work (p value = 0.459), There was no assossiation between work duration, work period and blood mercury levels with the incidence of liver disfunction in workers (p value = 0.148; 0.408 and 0.608). There was a relationship between blood mercury levels with SGPT as an indicator of liver dysfunction (p value = 0.042) Conclusion: Overall, the data did not provide strong evidence that mercury exposure associated with incidence of liver disfunction. Keywords: mercury exposure, liver disfunction, SGOT, SGPT, ALP }, issn = {2502-7085}, pages = {64--69} doi = {10.14710/jkli.12.1.64 - 69}, url = {https://ejournal.undip.ac.id/index.php/jkli/article/view/5962} }
Refworks Citation Data :
Background: Traditional gold mining activities that using WOA/ amalgamation can cause Hg emissions to the environment. The emissions could raise mercury poisoning in the environment and human. Liver as a major part of the metabolism and accumulation of Hg in the human body, so that Hg could lead to liver damage. In the previous research, Hg exposure in male rats caused hepatotoxicosis. Average blood mercury levels of workers was 53.5 μg/m3.
Objective: To determine the association between mercury (Hg) exposure and theoccurance of liver dysfunction on gold mine workers at Jendi Village, sub-district Selogiri, Wonogiri District.
Methods: It was a cross-sectional study, total sample were 41 workers. The data obtained from the results of laboratory tests of blood samples and the results of the interview respondents.Data would be analyzed using biavariate and multivariate statstic test.
Results:There was 41.16% of respondents were miners, processors as well as grates, the average of working period was 10 years, work duration 6 hours a day and 6 days a week. 97.56% of respondents have blood mercury levels above normal (U.S. EPA: 5.8 ppb). Elevated levels of SGOT experienced by 24.4% of respondents , SGPT 17.1% of respondents and ALP 58.8% of respondents or as much as 68.3% of respondents having liver disfunction. There was no difference incidence of liver dysfunction seen from the type of work (p value = 0.459), There was no assossiation between work duration, work period and blood mercury levels with the incidence of liver disfunction in workers (p value = 0.148; 0.408 and 0.608). There was a relationship between blood mercury levels with SGPT as an indicator of liver dysfunction (p value = 0.042)
Conclusion: Overall, the data did not provide strong evidence that mercury exposure associated with incidence of liver disfunction.
Keywords: mercury exposure, liver disfunction, SGOT, SGPT, ALP
Article Metrics:
Last update:
Last update: 2024-10-30 11:01:40
The Authors submitting a manuscript do so on the understanding that if accepted for publication, copyright of the article shall be assigned to Jurnal Kesehatan Lingkungan Indonesia (JKLI, p-ISSN: 1412-4939, e-ISSN:2502-7085) and Master Program of Environmental Health, Diponegoro University as the publisher of the journal. Copyright encompasses the rights to reproduce and deliver the article in all form and media, including reprints, photographs, microfilms, and any other similar reproductions, as well as translations.
JKLI journal and Master Program of Environmental Health, Diponegoro University, the Editors and the Advisory Editorial Board make every effort to ensure that no wrong or misleading data, opinions or statements be published in the journal. In any way, the contents of the articles and advertisements published in the JKLI journal are the sole and exclusive responsibility of their respective authors and advertisers.The Copyright Transfer Form can be downloaded here: [Copyright Transfer Form JKLI journal] The copyright form should be signed originally and send to the Editorial Office in the form of original mail or scanned document to jkli@live.undip.ac.id.
Jurnal Kesehatan Lingkungan Indonesia (e-ISSN: 2502-7085, p-ISSN: 1412-4939) is published by Master of Environmental Health, Faculty of Public Health, Universitas Diponegoro under Creative Commons Attribution-ShareAlike 4.0 International License.
View My Stats
